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There are several pathways that can explain each experiment that was performed and why it happened. The first experiment was to test to see how exercise affects BP as well as HR. It is hypothesized that immediately after exercise both subjects will have an increase in their arterial pressure as well as their heartrate will increase. This is because when a person is exercising, their bloodis pumping faster, which is causing the heart to beat faster and the pressure to rise. Subject #1 (well conditioned) baseline BP was 117/61 mmHg and had a HR of 66 bpm. Where as subject #2 (poor conditioned) baseline BP was 110/70 mmHg and HR had a HR of 80 bpm. Immediately after exercise both subjects BP and HR increased significantly. Subject #1’s BP went …show more content…
It is hypothesized that if subject #3 is reclining for 3 minutes, their arterial rate and heart rate will decrease because their body is relaxing and their heart is not doing any work. This drop is caused by the PNS system sending a signal to the cardioinhibitory center to lower heart rate. When the body is at rest, HR does not need to be increased. While the subject was sitting quietly, the baseline BP and HR was taken. BP was 105 mmHg and HR was 66 bpm. After 3 minutes of reclining, the subjects BP increased to 121/71 mmHg and HR decreased to 62 bpm. The cause of this could have been student error or the subject may have been uncomfortable during the experiment. After reclining for 3 minutes, the subject immediately stood up It is hypothesized that their arterial pressure and heart rate will stay the same because the subject is not moving therefore blood is being pooled in the lower extremities and not getting back to the heart. This will cause a decrease in HR and BP. But the body will sense this and activate the SNS response to release norepinephrine to increase BP and HR or to get it back to what BP and HR was initially. After standing for 3 minutes, the subjects BP went from 129/70 mmHg to 121/75 mmHg and HR went from 86 bpm to 80 bpm. As stated previously, blood was starting to pool into the legs. The results do not necessarily support the hypothesis because there was a decrease in BP and HR. This could be because the SNS had not been activated just yet because the body did not sense a change in BP and/or HR. The body will sense a decrease in returned blood and will act upon it by increasing BP and HR to get more blood coming to the heart. After 3 minutes, BP decreased from 129/70 mmHg to 121/75 mmHg and HR decreased from 86 bpm to 80 bpm. The results showed a decrease in BP as well as HR. As stated in the hypothesis it was expected to

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