There are
several pathways that can explain each experiment that was
performed and why it happened. The first experiment was to test to
see how exercise affects BP as well as HR. It is hypothesized that
immediately after exercise both subjects will have an increase in
their arterial pressure as well as their
heartrate will
increase. This is because when a person is exercising, their
bloodis pumping
faster, which is causing the heart to beat faster and the pressure
to rise. Subject #1 (well conditioned) baseline BP was 117/61 mmHg
and had a HR of 66 bpm. Where as subject #2 (poor conditioned)
baseline BP was 110/70 mmHg and HR had a HR of 80 bpm. Immediately
after exercise both subjects BP and HR increased significantly.
Subject #1s BP went
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It is
hypothesized that if subject #3 is reclining for 3 minutes, their
arterial rate and heart rate will decrease because their body is
relaxing and their heart is not doing any work. This drop is caused
by the PNS system sending a signal to the cardioinhibitory center
to lower heart rate. When the body is at rest, HR does not need to
be increased. While the subject was sitting quietly, the baseline
BP and HR was taken. BP was 105 mmHg and HR was 66 bpm. After 3
minutes of reclining, the subjects BP increased to 121/71 mmHg and
HR decreased to 62 bpm. The cause of this could have been student
error or the subject may have been uncomfortable during the
experiment. After reclining for 3 minutes, the subject immediately
stood up It is hypothesized that their arterial pressure and heart
rate will stay the same because the subject is not moving therefore
blood is being pooled in the lower extremities and not getting back
to the heart. This will cause a decrease in HR and BP. But the body
will sense this and activate the SNS response to release
norepinephrine to increase BP and HR or to get it back to what BP
and HR was initially. After standing for 3 minutes, the subjects BP
went from 129/70 mmHg to 121/75 mmHg and HR went from 86 bpm to 80
bpm. As stated previously, blood was starting to pool into the
legs. The results do not necessarily support the hypothesis because
there was a decrease in BP and HR. This could be because the SNS
had not been activated just yet because the body did not sense a
change in BP and/or HR. The body will sense a decrease in returned
blood and will act upon it by increasing BP and HR to get more
blood coming to the heart. After 3 minutes, BP decreased from
129/70 mmHg to 121/75 mmHg and HR decreased from 86 bpm to 80 bpm.
The results showed a decrease in BP as well as HR. As stated in the
hypothesis it was expected to
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