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Before the turn of the 21st century, physicians across the country were realizing that the possibility of being able to treat and cure virtually any microbial infection with the use of a single or a combination of antimicrobial medications was becoming more and more of a challenge. In the late 1960s and early 1970s, reports of pathogenic resistance to even the most potent antimicrobial medications of the time were being sent to the Centers for Disease Control. Among even the most dangerous pathogens that have developed and are developing drug resistance to one or many antimicrobials, Staphylococcus aureus (S. aureus) is perhaps a case in which the world is most familiar and of the greatest concern within the medical community due to its natural virulence-its ability to cause a multitude of life-threatening infections, and its above average ability to combat and adapt to a vast array of environmental conditions.

In the early 1940s, with the introduction of penicillin, the mortality rate among those with an S. aureus infection was dramatically improved. However, it is estimated that as early as 1942, only two years after the ‘miracle drug’ was available for public use, strains of S. aureus had already developed resistance to penicillin. More than 90% of S. aureus strains are resistant to penicillin today, thanks to the bacteria’s blaZ gene …show more content…
Aureus has taken even our most effective medications and rendered them useless even among the Staphylococcus genus. Researchers have developed yet more drugs, Quinupristin-dalfopristin, and linezolid, the actively fight against drug-resistant staphylococci. “Both agents are protein synthesis inhibitors with a Gram-positive spectrum. Quinupristin-dalfopristin retains bactericidal antistaphylococcal activity if the strain is susceptible to erythromycin and lincosamide. Linezolid is bacteriostatic. Cross-resistance has not been noted for linezolid, but at least one clinical isolate has developed resistance during

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